References
Ahmed, M., and Lyles, D.S. (1998). Effect of Vesicular Stomatitis Virus matrix protein on transcription directed by host RNA polymerases I, II, and III. J. Virol. 72, 8413-8419.
Ahmed, M., McKenzie, M.O., Puckett, S., Hojnacki, M., Poliąuin, L., and Lyles, D.S. (2003). Ability of the matrix protein of Vesicular Stomatitis Virus to suppress beta interferon gene expression is genetically correlated with the inhibition of host RNA and protein synthesis. J. Virol. 77,4646-4657.
Ahmed, M., Mitchell, L.M., Puckett, S., Brzoza-Lewis, K.L., Lyles, D.S., and Hiltbold, E.M. (2009). Vesicular stomatitis virus M protein mutant stimulates maturation of Toll-like receptor 7 (TLR7)-positive dendritic cells through TLR-dependent and -independent mechanisms. J. Virol. 83,2962-2975.
Albertini, A.A.V., Baquero, E., Ferlin, A., and Gaudin, Y. (2012). Molecular and celluiar aspects of Rhabdovirus entry. Viruses 4, 117-139.
Balachandran, S., and Barber, G.N. (2000). Vesicular stomatitis virus (VSV) therapy of tumors. IUBMB Life 50, 135-138.
Balachandran, S., and Barber, G.N. (2004). Defective translational control facilitates vesicular stomatitis virus oncolysis. Cancer Celi 5, 51-65.
Bateman, A., Bullough, F., Murphy, S., Emiliusen, L., Lavillette, D., Cosset, F.-L., Cattaneo, R., Russell, S.J., and Vile, R.G. (2000). Fusogenic membranę glycoproteins as a novel class of genes for the local and immune-mediated control of tumor growth. Cancer Res 60,1492-1497.
Blondel, D., Harmison, G.G., and Schubert, M. (1990). Role of matrix protein in cytopathogenesis of vesicular stomatitis virus. J. Virol. 64, 1716-1725.
Breitbach, C.J., Burkę, J., Jonker, D., Stephenson, J., Haas, A.R., Chow, L.Q.M., Nieva, J., Hwang, T.-H., Moon, A., Patt, R., et al (2011). Intravenous delivery of a multi-mechanistic cancer-targeted oncolytic poxvirus in humans. Naturę 477,99-102.
Burek, M., Maddika, S., Burek, C.J., Daniel, P.T., Schulze-Osthoff, K., and Los, M. (2005). Apoptin-induced celi death is modulated by Bcl-2 family members and is Apaf-l dependent. Oncogene 25, 2213-2222.