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Hypokalaemia

	*		— Alkalosis
	? Redistribution		— Insulin excess
	into cells		— Catecholamine fe-agonists
	\		— Hypokalaemic periodic paralysis
	r ~\ 9 I k intakp		r— Dietary l.v. therapy
	1
	(IN/i^oceił/o Ić 1 aoooc
Unne K1	CAk/^blYt) r\ lUboub		| Unne K
> 20-30 mmol/day 1			1 < 20-30 mmol/day
RENAL		GASTROINTESTINAL
With hypertension		With alkalosis
• Hyperatdosteronism		Vomiting
V- (incl. Conn’s syndrome)		Nasogastric aspiration
2* (with renal ischaemia)		With acidosis
• Other forms of mineralocorticoid		Diarrhoea
receptor activation		Laxative abuse
Cushings syndrome/ectopic ACTH		Villous adenoma of rectum
Corticosteroid therapy		Bowel obstruction/fistula
Apparent mineralocorticoid excess		Ureterosiamoidostomy
Liquorice/carbenoxolone
•    Liddles syndrome With normal-low blood pressure •    With alkalosis
Diuretic therapy (loop and thiazide) Barttefs and Gitelman’s syndromes
• With acidosis
Renal tubular acidosis (types 1 and 2) Carbonic anhydrase inhibitor therapy
• Wth variable pH
Post-obstructive diuresis
Recovery after acute tubular necrosis Mg depletion

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