3240962920

152

Mei, H., Chen, W., Dellinger, A., He, J., Wang, M., Yau, C., Srinivasan, S. et Be-renson, G. (2010). Principal-component-based multivariate regression for genetic association studies of metabolic syndrome components. BMC Genetics, 11( 1), 100.

Meng, Y. A., Yu, Y., Cupples, L. A. et Farrer, Lindsay A ANDLunetta, K. L. (2009). Performance of random forest when snps are in linkage diseąuilibrium. BMC bioinformatics.y (10 :78).

Mogi, M., Harada, M., Riederer, P., Narabayashi, H., Fujita, K. et Nagatsu, T. (1994). Tumor necrosis factor- a (tnf- a ) increases both in the brain and in the cerebrospinal fluid from parkinsonian patients. Neuroscience Letters, 1 £5(1-2), 208 - 210.

Morgenthaler, S. (2008). G6n6tique statistique. Springer Science &; Business Media.

Neter, J., Kutner, M. H., Nachtsheim, C. J. et Wasserman, W. (1996). Applied linear statistical models, volume 4. Irwin Chicago.

Nordborg, M. et Tavare, S. (2002). Linkage diseąuilibrium : What history has to tell us. Trends Genet., 18(2), 83-90.

011ivier, L. (2002). Eliments de genetiąue quantitative. Editions Quae.

011ivier, L. et al. (1971). L’heritabilite et sa mesure. Bulletins et Mćmoires de la Societe d'anthropologie de Paris, 7(2), 159-167.

Olsen, C. et Wagtmann, N. (2002). Identification and characterization of human dpp9, a novel homologue of dipeptidyl peptidase iv. Gene, 299(1), 185-193.

Ott, J. r. et Rabinowitz, D. (1999). A principal-components approach based on heritability for combining phenotype information. Human heredity, Ą9(2), 106— 111.

Patii, N., Berno, A. J., Hinds, D. A., Barrett, W. A., Doshi, J. M., Hacker, C. R., Kautzer, C. R., Lee, D. H., Marjoribanks, C., McDonough, D. P. et al (2001). Blocks of limited haplotype diversity revealed by high-resolution scanning of human chromosome 21. Science, 20^(5547), 1719-1723.

Perez-Palma, E., Bustos, B. I., Villaman, C. F., Alarcón, M. A., Avila, M. E., Ugarte, G. D., Reyes, A. E., Opazo, C., De Ferrari, G. V. et the AlzheimePs Disease Neuroimaging Initiative, t. N.-L. F. S. G. (2014). Overrepresentation of glutamate signaling in alzheimer’s disease : Network-based pathway enrichment using meta-analysis of genome-wide association studies. PLos one, 0(4), e95413.