Rrsponse to txeras»
Anticipation of exercise stimulates cardioregulatory centers, increasing heart ratę
nerve (X)
Sympathctic inhibition by baroreceptor mechanism is overwhelmed by generalized slimulation of sympathetics
Sympathetic
Sympathctic
' .rei.r
nerves
Catecholamine output by suprarcnal medul-lae promoted by sympathetic stimulation
Baroreceptors stimulated by hse in blood pressure; fali in blood pressure de creases tonie sympathctic inhibition
Sympathetic nerve stim-ulntion and circulating catecholamines, plus rełative decrease in va* gal tonę, accelerate SA node discharge ratę
Increased ratę of contraction
Skin: vasoconstriction at first. then dilation for heat dissipation
Increased venous return due to action of muscle pump and respiratory movemenls "
Sympathctic nerves and circulating catecholamine dilate coronary arteries iirv crease O 2 supply and mc-tabolite removal) and ad direclly on heart muscle. accelerating myocardial metabolism
Liver and splanchnic beds blood llow diminishes
Increased
Kidneys: blood flow diminishes
Muscle: inilial compression followed by marked vasodilation due to relcase of mctabolites and circulating epinephrine
Figurę 4.21 Circulatory Response to Exercise ___._
This figurp summarizes the integrated neural and Chemical effects of effects are mediated locally by the release of va$oactive metabf »!■•—
exercise on the cardiovascular system. Neural effects are mediated and the effects of circulating catechołamines (e.g.. epinepłuinc i centrally (by the autonomie nervous system), whereas the Chemical
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