IgA nephropathy
Minimal
change
nephropathy
Diabetic
nephropathy
Post-streptococcal Anti-GBM MCGN glomerulonephritis disease
FSGS
Membranous
nephropathy
Amyloid
Small-
vessel
vasculitis
Nephrotic
Mechanism
• Injury to podocytes
• Changed architecture
Scarring
Deposition of matrix or other elements
Haematuria
Proteinuria
Mechanism
• Inflammation
• Reactive celi proliferation
• Breaks in GBM
• Crescent formation
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