DePace, N. (1912). Sulla Scomparsa di un enorme cancro begetante del callo dellutero senza cura chirurgica [Italian]. Ginecologia 9.
DePolo, N.J., Reed, J.D., Sheridan, P.L., Townsend, K., Sauter, S.L, Jolly, D.J., and Dubensky Jr., T.W. (2000). VSV-G pseudotyped lentiviral vector particles produced in human cells are inactivated by human serum. Mol. Ther. 2, 218-222.
Der, S.D., Zhou, A., Williams, B.R.G., and Silverman, R.H. (1998). Identification of genes differentially regulated by interferon a, P, or y using oligonucleotide arrays. Proceedings of the National Academy of Sciences 95,15623-15628.
Derynck, R., Akhurst, R.J., and Balmain, A. (2001). TGF-beta signaling m tumor suppression and cancer progression. Nat. Genet. 29,117-129.
Derynck, R., Goeddel, D.V., Ullrich, A., Gutterman, J.U., Williams, R.D., Bringman, T.S., and Berger, W.H. (1987). Synthesis of messenger RN As for trans forming growth factors a and p and the epidermal growth factor receptor by human tumors. Cancer Res 47, 707-712.
Desforges, M., Charron, J., Berard, S., Beausoleil, S., Stojdl, D.F., Despars, G., Laverdiere, B., Bell, J.C., Talbot, P.J., Stanners, C.P., and Poliquin, L. (2001). Different host-cell shutoff strategies related to the matrix protein lead to persistence of vesicular stomatitis virus mutants on fibroblast cells. Virus Res. 76, 87-102.
Desforges, M., Despars, G., Berard, S., Gosselin, M., McKenzie, M.O., Lyles, D.S., Talbot, P.J., and Poliąuin, L. (2002). Matrix protein mutations contribute to inefficient induction of apoptosis leading to persistent infection of human neural cells by vesicular stomatitis virus. Virology 295, 63-73.
Diaz, R.M., Galivo, F., Kottke, T., Wongthida, P., Qiao, J., Thompson, J., Valdes, M., Barber, G., and Vile, R.G. (2007). Oncolytic immunovirotherapy for melanoma using vesicular stomatitis virus. Cancer Res. 67, 2840-2848.
Dickson, R.B., Kasid, A., Huff, K.K., Bates, S.E., Knabbe, C., Bronzert, D., Gelmann, E.P., and Lippman, M.E. (1987). Activation of growth factor secretion in tumorigenic States of breast cancer induced by 17 beta-estradiol or v-Ha-ras oncogene Proceedings of the National Academy of Sciences 84, 837-841.